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Deletions of AXIN1, a component of the WNT/wingless pathway, in sporadic medulloblastomas

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Despite the many studies which have measured GSH levels in various models of MASLD/MASH, results have been very inconsistent (Table S7)

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Their actions on multiple pathways, including potentially CysLTR2-expressing vagal and nonpeptidergic nociceptors in addition to the induction of epithelial GDF15, may explain the profound lack of avoidance seen following LTC4S deficiency, which was only partially recapitulated by deficiency of CysLTR2