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196 Stress exposure is a major pathogenic factor in disease models and can increase Ca 2+ -dependent release of ATP from neurons, which causes excitotoxicity

Development of IR is coupled with reduced glucose uptake in insulin-sensitive tissues, especially hepatic and skeletal muscle

neurorehabilitation models suggest amplification of exercise-driven plasticity through BDNF/TrkB, SIRT1, and related pathways

Although the National Institute of Health (NIH) still claims that GSH deficiency is very rare, there is growing number of reputable sources that state quite the opposite