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For example, whatever the radiation quality, an increase in the endogenous GSH pool may inhibit sister chromatid exchange [22], increase exchange aberrations [19], or reduce the frequency of aberrant metaphases [18]

Anatomically, portal hypertension can be classified 17 as (a) prehepatic (i.e., portal vein), (b) intrahepatic or (c) posthepatic (i.e., hepatic veins, thoracic caudal vena cava, or heart)

When ROS production exceeds the capacity of the cells antioxidant systems it impairs OXPHOS complex and further promotes ROS production (168, 169)

The evolution from daily injections to weekly injections to oral tablets to patches reflects a clear industry trajectory toward making these therapies as frictionless as possible

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