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In conclusion, glutathiones whitening effect acts on the whole process of melanin production

Th17 cells produce IL-17, which aggravates endothelial damage and increases plaque instability (10)

Conclusion: These data strongly suggest that the dysbiotic microflora characterized by a decrease in butyrate producing bacteria plays a causal role in MDB formation and pathogenesis of AAH

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It functions as a direct antioxidant through its thiol group, a substrate for glutathione peroxidase and glutathione S-transferase enzymes, and a regulator of protein function through S-glutathionylation