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Description

Increased plasma FFAs reduce glucose uptake stimulated by insulin, but the decrease of lipid content improves insulin activities in skeletal muscles, adipocytes, and the livers [4, 5]

IGFBPs can also act to guide IGF-1 to specific tissues, or even to inhibit or potentiate IGF-1 actions by acting as an independent substrate for the IGF-1R and/or other specific membrane, intracellular or nuclear receptors [9395]