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doi: 10.1016/j.jaut.2020.102510 116 NaYRKimSWSeokSH

The biosynthesis of glutathione (GSH) and the normal function of GPX4 are central to the control of ferroptosis, and the inhibition of GPX4 can increase cellular ferroptosis sensitivity [6, 7]

Once inflammation subsides, re-epithelialization begins, typically around 23 days after injury, as fibroblasts infiltrate the wound site, initiating collagen and extracellular matrix (ECM) deposition to create a scaffold for tissue repair