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The strongest direct human data on CJC-1295 involve the DAC-modified long-acting analog , not the No DAC version. For CJC-1295 without DAC, the mechanistic rationale is based on the broader GHRH (1-29), sermorelin, and Modified GRF literature. That mechanism is biologically well grounded, but the evidence should still be described carefully

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CJC 1295 binds to GHRH receptors, initiating steady GH release, while Ipamorelin binds to ghrelin receptors, inducing a rapid burst of GH