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In all, the data demonstrate that supraphysiologic increases in mitochondrial ubidecarenone levels is sufficient to alter redox homeostasis as well as promote site-specific ROS production at the level of the mitochondria at Complex II and mGPDH

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Accumulating evidence has shown that MAT2A is overexpressed in various tumors, including hepatic cancer 10,11 , breast cancer 12 , gastric cancer 13 , lung cancer 14 and glioma 15

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