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Hydrolysis of irinotecan and its oxidative metabolites, 7-ethyl-10-[4-N-(5-aminopentanoic acid)-1-piperidino] carbonyloxycamptothecin and 7-ethyl-10-[4-(1-piperidino)-1-amino]-carbonyloxycamptothecin, by human carboxylesterases CES1A1, CES2, and a newly expressed carboxylesterase isoenzyme, CES3

Whilst the mouse model has been invaluable in answering many questions associated with mitochondrial disease etiology and pathogenesis, it seems to pose some challenges just like the other models described previously

H., Mouro A., Buday K., Sato M., Wanninger J., Vignane T., Mohana V., Rehberg M., Flatley A., Schepers A., Kurz A., White D., Sauer M., Sattler M., Tate E

Therefore, a variety of xenobiotic glucuronides must be used to examine the activities of different gut bacterial GUSs to better understand the individual roles of the GUSs in GUS-induced pathology