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Studies have demonstrated that PGE 2 and its EP receptors are implicated in promoting carcinogenesis in different types of cancer (Wang and Klein, 2007)

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The relatively small number of RCTs available for each outcome measure (e.g., blood pressure, arterial stiffness, muscle function) precluded a quantitative meta-analysis, limiting the ability to draw robust statistical conclusions

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These findings indicate that even though ethanol may not be required for TNBS-induced damage, this vehicle enhanced the biological action of TNBS in agreement with previous results (Morris et al, 1989), presumably by acting as a barrier breaker and facilitating its interaction with cellular GSH stores (Table 1)