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[66] The trial's primary endpoint was the incidence of treatment-emergent adverse events (TEAEs)

In agreement with this, we observed the highest expression of PrP in cholangiocarcinoma nodes in the liver of Atp7b / mice, whereas PrP depletion almost completely prevented tumor growth

in keeping with 17-hydroxysteroid dehydrogenase activity which complements 5-reductase activity and DHT synthesis

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Bioavailability studies show significantly higher relative bioavailability compared with regular GSH, with a longer time to peak plasma concentration and a longer duration of action