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Extracellular matrix degradation in liver fibrosis: Biochemistry and regulation

Cancer Res 79(18):45574567

243 In AML, the level of H3K36me3 changes most dramatically before and after Met deprivation, and inhibition of STED2, the H3K36-specific methyltransferase, can reproduce most of the cytotoxic phenotypes produced by Met deprivation