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The proposed mechanism for hyperhomocysteinaemia on inducing endothelial dysfunction and thus atherosclerosis includes homocysteine-induced endoplasmic reticulum stress, oxidative stress, and proinflammatory response.48 Animal models of hyperhomocysteinaemia have confirmed the causal relationship between hyperhomocysteinaemia and the development of endothelial dysfunction and accelerated atherosclerosis.48 Although meta-analyses have shown reduction of cardiovascular risk with reduction of homocysteine levels,46 47 vitamin supplementation (with vitamin B 6 , vitamin B 12 , and folic acid) to lower homocysteine in the body may not be transformed into clinically beneficial vascular outcomes

For Healthcare Professionals Vitamin B12, part of the vitamin B-complex family, refers to the group of compounds that consists of cyanocobalamin, hydroxycobalamin, and related substances (1)

Butterworth RF

This solution numbs the area and minimizes bleeding

Spearman's rank correlation was used to analyze pairwise correlations between multiparameter MR metrics and mGSV