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Description

Role of uremic toxins in vascular inflammation associated with chronic kidney disease

Methods Cell

Endothelial cell (EC) activation and dysfunction, activation of macrophage (Figure 4)

Functional Outcomes of Caspase-4 and -5 Activation PyroptosisThe Inflammatory Cell Death Process Although the active site of inflammatory caspases is highly conserved, inflammatory caspases can have distinct substrate specificities

In ECA109 esophageal cancer cells, ISA significantly increases the expression of death receptor 5 (DR5) and reduces the cleavage of procaspase-10 (due to activation of caspase-10) (Lu et al., 2018)