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This mechanism reduces mitochondrial reactive oxygen species production while simultaneously improving bioenergetic output a dual action that distinguishes it from conventional antioxidants

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For instance, the virulence-associated antigen, ESAT-6, induces the M1 phenotype in early Mtb infection and later causes the transition to M2 (Refai et al., 2018)

Its primary mechanisms are: Actin regulation critical for cell migration and tissue repair Anti-inflammatory signalling through the NF-B pathway19 Angiogenesis promoting new vessel formation in damaged tissue20 Cardioprotection demonstrating direct myocardial repair properties in animal models21 TB-500 Cardiovascular Long COVID: The Strongest Use Case Long COVID cardiovascular sequelae are well-documented: Myocarditis and pericarditis in a subset of patients POTS (postural orthostatic tachycardia syndrome) Reduced VO max and exercise intolerance Microclotting and endothelial damage TB-500's most compelling evidence base is cardiovascular