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Figure 9 4 Discussion Glutamine, an amino acid needed for protein synthesis and for cellular proliferation, is converted to glutamate by glutaminase, and the disruption of cancer metabolism through glutamine inhibition has been an attractive target for solid and hematological tumor therapies for many years

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The longer effective tissue exposure is consistent with the actin-sequestration mechanism, which depends on intracellular accumulation rather than transient extracellular signaling

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Bcl2-A1 interacts with pro-caspase-3: implications for amyotrophic lateral sclerosis