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A question that remains to be answered is whether GSH catabolism occurs primarily in tumours or in non-tumorigenic tissue (for example, kidney)

BACH1 facilitates the recruitment of CARM1 to the promoters of VSMC marker genes, thereby enhancing their transcription through the augmentation of H3R17me2 modification, thus playing a role in the differentiation of VSMCs following mesoderm induction

There is also increasing evidence that deregulation of GSH synthesis contributes to the pathogenesis of diseases such as diabetes mellitus, pulmonary and hepatic fibrosis, alcoholic liver disease, cholestatic liver injury, endotoxemia, and drug-resistant tumors cells [10]

Ascorbic acid suppresses house dust mite-induced expression of interleukin-8 in human respiratory epithelial cells

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