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Miyake, K., Ohmori, K., Ishii, A

To restore drug sensitivity and further enhance antitumor effects, the hybrid siRNA anti-Survivin and anti-MDR1 (siSM) were treated as a model therapeutics via a PGS delivery system, resulting in a knockdown of Survivin and MDR1 and further sensitizing cancer cells to the drug cisplatin (DDP), PGS complexes were injected intravenously into a lung in situ tumor model, and it was found that PGS/siSM relatively reduced the growth rate of tumors, while simultaneous administration of PGS/siSM and DDP enhanced this effect ( in vivo and in vitro , down-regulating intratumoral induction of CD47 gene, mRNA and protein functioning as evading phagocytic clearance through binding to the immune receptor SIRP

The mechanism of ferroptosis regulating oxidative stress in ischemic stroke and the regulation mechanism of natural pharmacological active components

29 and Supplementary Figs