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This can become of use for further research in the future

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DOI: 10.1111/nmo.14264

The evolution of metazoan TrxRs from Gsr involved acquisition of a carboxy (C)-terminal extension to the enzyme that encodes the active site responsible for reducing Trx-disulfides [48, 51, 54]

Not all conditional GPX4 mouse models produce spontaneous disease phenotypes on a normal diet, although a vitamin E-depleted diet accelerates the pathogenic phenotype of cell death [31]