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TCIs exert their anti-inflammatory effects by suppressing calcineurin-dependent T-cell activation, thereby preventing the release of pro-inflammatory cytokines and mediators

In experiments with a middle cerebral artery occlusion model induced by nylon monofilament insertion and rat cortical neurons (RN-c MZ-7885) subjected to oxygen and glucose deprivation/reperfusion, catalpol activates the NRF1/K (lysine) acetyltransferase 2A (KAT2A)/methyltransferase like 3 (METTL3) axis and downregulates Beclin-1 expression, thereby relieving neuronal injury and excessive autophagy after cerebral ischemia (Liu K

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PMID: 38363684 Cell Rep CDKL5 deficiency in adult glutamatergic neurons alters synaptic activity and causes spontaneous seizures via TrkB signaling