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The reason why cells can generally sustain their NAD + levels and, therefore, their viability, is because they can salvage NAD + via multiple endogenous and exogenous precursors

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These changes in the NH 3 /NH 4 + equilibrium drive measurable potential shifts in potentiometric sensors, enabling quantification through ammonium-selective electrodes [198]

GO enrichment analysis suggested that the autophagy-related upregulated DEGs were enriched in pathways closely related to the cell cycle, cell proliferation, antioxidant stress, and energy metabolism functions, whereas the downregulated genes were enriched in pathways related to infection, immune defense, and chemical signaling

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