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2) Cell type- and clinical phenotype-specific regulatory blank spots: Most research focuses on human mesangial cells, but the cell type-specific contribution to IgA nephropathy-related ferroptosis is severely understudied

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Building trust and deepening connection is a hard-won struggle, and there have been many decades of experience to inform our work going forward

V.GettingS.et al (2022)

Ischemic cells compensate for reduced oxygen delivery by increasing anaerobic glycolysis, leading to acidosis as well as glucose derivatives that promote signaling related to oxidative stress and inflammation [38, 57, 62]