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Accordingly, A-exposed astrocytes stimulate an increase in endothelial TfR1 and DMT1 expression in patients brain regions vulnerable to AD pathology, such as the hippocampus and cortex, thereby potentially increasing transferrin-mediated iron uptake by endothelial cells [105]

Since these granules are formed only at the promyelocyte stage, their depletion under chronic demand leads to neutrophil exhaustion and impaired cortisol delivery

In contrast, GSH depletion drives M1 polarization, resulting in heightened secretion of pro-inflammatory cytokines, including TNF-, IL-6, and IL-1 (Kwon et al., 2019)

Michael B, Coyne DW, Fishbane S, Folkert V, Lynn R, Nissenson AR, et al

While the research discussed in this review focuses on the role of ferroptosis in cell death of tissues directly exposed to IR, there is evidence that ferroptosis plays a role in other systemic side effects of radiotherapy