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2017 , 5 , e13281

This article was created with the assistance of Generative AI and has undergone editorial review before publishing

From a GABAergic perspective, the neuroprotective role of CX3CL1 could be attributed in part to enhanced inhibitory currents and suppressed AMPA receptor function during the acute excitotoxic phase of ischemia (Cipriani et al., Studies on both physiological and pathological conditions, such as epilepsy, demonstrate that the CX3CR1 pathway may regulate the number of post-synaptic GABA A receptors or their sensitivity to GABA and thus their subunit composition (Heinisch and Kirby, 2009

Mitochondrial defects arise from nucleoside/nucleotide reverse transcriptase inhibitors in neurons: Potential contribution to HIV-associated neurocognitive disorders

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