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coli enhance the disease phenotype in a hSOD1-G93A mouse model of ALS

Thiolation of low-density lipoprotein by homocysteine thiolactone causes increased aggregation and altered interaction with cultured macrophages

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More importantly, a growing body of research reveals that these hepatoprotective effects do not stem from direct liver-targeted actions of TCM components but are achieved through an indirect pharmacological model: TCM acts as a 'modulator' to reshape the GM structure, which subsequently alters the profile and levels of microbiota-derived intermediary substances (e.g., metabolites such as SCFAs, BAs, TMAO, LPS, etc.)