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High insulin levels in the post-prandial state decrease hepatic VLDL production, favoring chylomicron-mediated delivery of dietary lipids to the periphery [111], but the selective hepatic insulin resistance in patients with NAFLD allows insulin to stimulate DNL without inhibiting VLDL production [118]

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This paper presents human studies that analyse the role of GLP1 release in the glucose-lowering effect of metformin

Labetalol, a mixed / blocker, is a third-line agent, whereas nonselective -blockers such as propranolol ( Class III ) are contraindicated

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