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Cytokine networks in the pathogenesis of rheumatoid arthritis

In macrophages, reorganization of the actin cytoskeleton necessary for chemotaxis and phagocytosis requires MsrB1-dependent reduction of methionine-R-sulfoxide residues within actin (27)

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M1 macrophages are less prone to ferroptosis and maintain proinflammatory activity because of the inhibition of arachidonate 15-lipoxygenase (ALOX15), a key enzyme in ferroptosis, by nitric oxide (NO) produced via inducible nitric oxide synthase (iNOS) [58,59,60]