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Conversely, the inhibition of the de novo synthesis of GSH dampens intracellular antioxidant defence mediated by this system and increases oxidative stress (Reference Lian, Gnanaprakasam and Wang28)

Furthermore, important questions remain, including the long-term durability of weight loss and its maintenance after treatment discontinuation, as well as its impact on major cardiovascular outcomes

Through these mechanisms, phytochemicals improve mitochondrial function, inhibit abnormal cell survival signaling, induce apoptosis in ectopic lesions, and contribute to the restoration of endometrial homeostasis

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Hypercholesterolemia impairs endothelium-dependent relaxations in common carotid arteries of apolipoprotein e-deficient mice