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This schematic highlights how curcumin disrupts key inflammatory cascades such as the IKK/NF-B pathway, shifts immune cell phenotypes toward anti-inflammatory states, suppresses the expression of pro-fibrotic factors such as TGF- and CTGF, and promotes the apoptosis of inflammatory cells

Here we review the evidence for white matter abnormalities in AD with a focus on myelin and oligodendrocytes, the only source of myelination in the central nervous system, and discuss the relationship between white matter changes and the hallmarks of Alzheimers disease

We then established a putative gene signature (comprising gclc , gls , gpx4 , gss , slc25a39 , and e2f1 ) of GSH metabolism (GMGS) in association with therapeutic response

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Moreover, a unique point mutation of PSMB5 (gene encoding the proteasome 5 subunit) alleviates fatal ER stress, promoting bortezomib resistance in MM cells (122)