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Naturally found in human plasma, this complex combines three vital amino acids: glycine, histidine, and lysine, with copper ions to act as a "signal peptide." In the ecosystem of the scalp, GHK-Cu functions as a biological architect, sending specific instructions to cells to initiate repair, dampen inflammation, and jumpstart the hair production cycle

Preclinical research has also observed downregulation of pro-inflammatory cytokines including TNF-, IL-6, and IL-1, alongside modulation of NF-B signalling pathways positioning GHK-Cu as a subject of interest in inflammatory resolution research

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This happens through the induction of telomerase enzyme activity, resulting in the extension of telomere length

Interestingly, NF-B p65 and caspase-3 were significantly inhibited by trifluoperazine, a strong calmodulin antagonist, suggesting that the cardioprotective effect conferred by trifluoperazine involves inhibition of NF-B and apoptosis