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& He, X.-X

2008, doi: 10.1016/j.abb.2008.01.028

That is why its effect in studies extends far beyond the gastrointestinal tract, where it was originally discovered

whether contrast-induced oxidative stress acts as a direct trigger, or if other intermediates (e.g., endoplasmic reticulum stress and mitochondrial dysfunction) are involved, requires definitive validation

Depletion of cystine/cysteine reduces intracellular GSH levels and GPX4 activity within the cystine/cysteine-GSH-GPX4 axis, thus causing ferroptosis (18,99)