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PICALM regulates cathepsin D processing and lysosomal function

However, recent reports indicate that the different KRAS mutants exhibit distinct biological characteristics and have varying impacts on patients [7]

The complex pathophysiology of NASH, and a predicted prevalence of 35% of the adult population worldwide, has prompted drug development programs aimed at multiple targets across all stages of the disease

et al., 2018) revealed that AMPK-mediated phosphorylation of BECN1 at Ser 90/93/96 is essential for lipid peroxidation

Further, these manifestations may not be progressive, which makes diagnosis and management challenging