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The SP@M treatment led to a significant decrease in the fluorescence intensity of CD86 (the phenotypic marker of M1-like macrophages) and an increase in the intensity of CD206 (the phenotypic marker of M2-like macrophages), indicating the remission of pro-inflammatory microenvironment (Supplementary Fig

GLS1i reduced fibrosis markers in an acute liver fibrosis model (single injection of CCl4) 4 and a bleomycin model (BleoM) of idiopathic pulmonary fibrosis (IPF) 5