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A promoter impaired cup1-tt allele also mediates resistance to caffeine, consistent with cup1 + repression being the source of resistance phenotypes in the UR2 epimutant (Torres-Garcia et al, 2020b)

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In one rat study, BPC-157 administration promoted tendon fibroblast outgrowth, cell survival under stress, and cell migration through the FAK-paxillin signaling pathway