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In further support of this model, glucose starvation-induced cell death and redox defects in SLC7A11 high cancer cells can be substantially blocked by treatments that restore NADPH levels and prevent cystine accumulation, such as treatment with 2DG (which inhibits glycolysis but can still be shunted into the PPP to generate NADPH) (Liu et al., 2020), suggesting that it is cystine accumulation and/or NADPH depletion resulting from cystine uptake that causes the cell death in SLC7A11 high cancer cells under glucose starvation

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Bacterial antigens are typically screened primarily as recombinant proteins, whereas viral antigens can be analyzed using genetic vaccinations

These results suggest that DHA metabolites rather than DHA itself may account for the antitumor action of DHA