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Disrupting the putative binding sites for -globin repressors like BCL11A to induce HbF production will be an attractive therapeutic strategy for both -thalassemic and SCD patients (Masuda et al., 2016

albicans has shown that candidalysin stimulates the production of IL-1 through a CARD9-dependent mechanism, thereby leading to neutrophil recruitment mediated by the chemokine CXCL1 122

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It is a key distinction from growth hormone secretagogues or GHRH analogues that require functional pituitaries to work