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In this respect, it should be noted that many of these diet-related metabolites were microbial-transformed compounds (e.g., phenolic acids, valerolactones)

This contrasts with the aforementioned direct protective effects targeting the brain or spinal cord in neurological diseases, yet shares a consistent mechanistic core: upregulating Nrf2 at central nervous system sites can significantly influence the pathological progression of systemic or multi-system diseases

These values represent reported laboratory handling only and are provided strictly for research use, not human use

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Nevertheless, genetic variation only explains a minor part of interindividual variability, suggesting regulation by nuclear receptors, PAPS-cofactor availability and inhibition by various drugs and environmental compounds could be major determinants of variability in SULT activity 15