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Therefore, FOXO4-DRI's targeted mechanism suggests a potential research outcome by facilitating the removal of senescent cells, which may contribute to tissue rejuvenation and improved organ function while still preserving the critical antioxidant defenses mediated by FOXO4

Acetylcysteine (MUCOMYST) Aflatoxin B 1 Arsenic Atropine Benzo[a]pyrene Cadmium Chromium Deferasirox (EXJADE) Deferoxamine (DESFERAL) Dimercaprol Dimercaptosuccinic acid (DMSA), succimer (CHEMET) Diphenhydramine (BENEDRYL) EDTA CaNa 2 Ethanol Flumazenil Fomepizole Lead Mercury Naloxone (NARCAN) Penicillamine Physostigmine Pralidoxime chloride (2-PAM) Sodium 2,3-dimercaptopropane sulfonate (DMPS)not approved by FDA, but approved for use in Germany a A complete list of antidotes is shown in Table 3-1

The higher concentration of 3,4-diOH-TAM oxidized a greater amount of NADH

GR activity was expressed as nM NADPH oxidation min 1 mg 1 protein

Br J Pharmacol 2009