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The mechanism of TIGAR inhibiting neuroinflammation may be related to the clearance of ROS, because ROS is a powerful promoter of neuroinflammation (Giffard and Swanson, 2005)

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It may be speculated that partial glutathionylation of class I GRXs even under the high GSH/GSSG ratio present in living cells provides a reservoir of glutathionylated GRXs to facilitate rapid glutathionylation of reactive cysteines