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The use of these proteins would be based on their expected biological activity: DSIP as the active peptide expected to induce sleep, and CBBBP to facilitate entry into brain

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Therefore, addressing homocysteine levels and supporting methylation can help mitigate long-term health risks

Clinical ASD arises from diverse etiologies including de novo mutations (e.g., SHANK3, CHD8), copy number variations, and polygenic risk factors [44]

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