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10.1016/j.nbd.2011.04.025 Neurobiol

In addition, they found that the liver inflammation of T2DM rats significantly improved, due to the expression of myeloid differentiation factor 88 (MyD88), TNF receptor-associated factor 6 (TRAF6), toll-like receptor 4 (TLR4), an inhibitor of NF-B alpha (I), p-I, and NF-B were downregulated (128)

In summary, metabolic reprogramming in trained immunity involves the coordinated regulation of glucose, amino acid, and lipid metabolism

Peterson FC et al (2011) Orphan macrodomain protein (Human C6orf130) is an O-Acyl-ADP-ribose deacylase

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