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The presence of Faecalibacterium is a sign of successful ustekinumab therapy in anti-tumor necrosis factor-alpha (anti-TNF-) refractory patients with CD (93)

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Therefore, in the unphosphorylated state, enhanced flexibility in the SH3SH2 linker appears to promote arr1 accessibility by transiently exposing SH3 binding interface to arr1 (Fig

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Geladaris, A

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