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In contrast, iNOS, expressed in response to inflammation, produces micromolar quantities of NO (Nathan and Xie, 1994)

These findings, together with various oligomerization states of VP35 in different filoviruses 16 , imply that the differences in VP35 dynamics among the filoviruses may dictate the potential divergent mechanisms of replication and pathogenesis during viral propagation

In Fmr1 knockout mice, GABAergic inhibitory function in the CA1 region of the hippocampus was impaired, as evidenced by reduced amplitude and frequency of eIPSCs, spontaneous IPSCs, and miniature IPSCs, along with significantly decreased expression of 2, 1, and GABAA receptor subunits in the hippocampus, which is associated with behavioral and cognitive deficits

The details of the kits are listed in Supplementary Table 6