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One approach in creating a mitochondria-targeted AT is to bind an antioxidant molecule (e.g., SOD, thiol, vitamin E, BHT, ebselen, CoQ, or various spin traps, such as TEMPO) with a hydrocarbon chain of varying length to the phosphorus of the lipophilic compound triphenylphosphonium (TPP) such as mitoquinol (MitoQ 10

12 Now we recognize that ROS can be generated through mechanisms such as the mitochondrial electron transport chain, the endoplasmic reticulum, and NOX

showed that the accumulation of succinate, that is oxidized by complex II (succinate dehydrogenase), results in mtROS production, seemingly from RET at complex I ( 2.2.2 Monoamine Oxidases A relevant mitochondrial-specific source of ROS is monoamine oxidase (MAO), although its impact on inflammation has been quite overlooked

Antacids and proton pump inhibitors such as Prilosec, Protonix, Prevacid, Aciphex, Zantac and Pepcid also can lead to B12 deficiency, because they reduce the stomach acid necessary to release the B12 from food

efflux channel