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This occurs through enzymes such as SIRT1, which can interact with molecular clock proteins like CLOCK and PER2, affecting their acetylation status (Nakahata et al., 2008), or through the protein kinase mTOR, whose activity exhibits rhythmicity (Khapre et al., 2014b) and plays a key role in both aging and tumorigenesis (Weichhart, 2018

One challenge that tends to occur in autistic children is difficulty with metabolic processes, specifically methylation and oxidative stress 2,3