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Keywords Reactive nitrogen species, reactive oxygen species, endoplasmic reticulum stress, unfolded protein response, drug-induced liver injury Introduction Drug-induced liver injury (DILI) plays an essential role both in the clinic and in drug development [1]

Other mechanisms that regulate the intracellular iron abundance, such as the exportation of iron via various multivesicular bodies and exosomes carrying ferroportin or prominin2-mediated ferritin, can also affect the cellular sensitivity to ferroptosis, as they deplete the cellular iron and accelerate the lipid peroxidation (53) ( Figure 1 )

The interplay between microglial function and systemic metabolic states is gaining increasing attention

Comp Hepatol 2(1):121

2020;12:15 Salardini E, ODell RS, Tchorz E, Nabulsi NB, Huang Y, Carson RE, et al