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By inhibiting NNMT, 5-Amino-1MQ may spare nicotinamide for NAD+ synthesis via the salvage pathway, thereby activating SIRT1 (Sirtuin 1) pathways associated with mitochondrial biogenesis, fat oxidation, and metabolic flexibility [3]

On top of this, an enzyme called carnitine palmitoyltransferase-1 (CPT-1) is freed from additional inhibition, potentially shuttling more fatty acids into mitochondria for burning rather than storage

Copper peptides (GHK-Cu) are complexes composed of glycyl-L-histidyl-l-lysine tripeptides (GHK) and divalent copper [[16], [17], [18]]

Patients may require another treatment, 6-10 weeks later, depending on the severity of the area being treated