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249 The key molecular event of KD treatment in promoting immunotherapy is the activation of AMPK, which phosphorylates PD-L1, resulting in its disrupted interaction with CMTM4 and subsequent PD-L1 degradation, which phosphorylates EZH2, resulting in improper PRC2 function and subsequently enhanced expression of IFN and antigen-presentation gene

These findings are in agreement with previous report [67]

"Implication of interleukin family in cancer pathogenesis and treatment." Cancers 13.5 (2021): 1016

Bibcode:2000AcChR..33...94L

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