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doi: 10.1016/j.jcrc.2013.12.003

Notably, specific ferroptosis inhibitors show efficacy not only in RF models but also in rodent models of kidney and liver cancer (195), underscoring ferroptosis in TECs as a therapeutically tractable node in fibrogenesis

1B) [39]

This highlighted the importance of implementing an antioxidant system that could mitigate the negative charge imbalance caused by overexpression of ROS

Chronic treatment with PARP inhibitors increased NAD + levels and mitochondrial respiration in 10-week-old mice and triggered UPR mt and increased mitochondrial respiration and lifespan in worms 109,257