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Importantly, the activation of AMPK is identified as an inhibitory mechanism against Gal-3-induced PASMC proliferation by targeting YAP/FOXM1/Cyclin D pathway

Activation of MEK1/2 activates MAPK, which is the downstream molecule in the signaling pathway and controls the final cell response to stress [165, 172, 173]

These findings suggest that differences in P-gp inhibitory activity among ARBs may influence bleeding risk in clinical practice when co-administered with DOACs

Protective role of PGC-1alpha in diabetic nephropathy is associated with the inhibition of ROS through mitochondrial dynamic remodeling

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